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Preclinical data show enhanced antioxidant capacity through increased intracellular glutathione, activation of the Nrf2 pathway, & elevated NQO1 expressionkey mechanisms in cellular defense against oxidative stress
PPAR and PPAR/ are mainly responsible for regulation of fatty acid degradation, whereas PPAR takes part in lipid storage, regulates adipocyte differentiation, and enhances adipogenesis and insulin sensitivity [100,101,102,103,104]
Several enzymes involved in the glutathione pathway, such as -glutamylcysteine synthetase (GCLC), glutathione synthetase (GSS), glutathione peroxidase 4 (GPX4), and glutathione reductase (GSR), are regulated, at least in part, by Nrf2 through its activation of the Nrf2-antioxidant response element
Dr Kapoor explains the symptoms as follows: 1
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