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Specifically for Keratosis Pilaris, genes BRAF, MEK1, MEK2, and KRAS are implicated in cardio-facio-cutaneous (CFC) syndrome, where the predominant features from these gene mutations are Keratosis Pilaris and Ulerythema ophryogenes among other epidermal abnormalities

Although increased passive stiffness is commonly attributed to myocardial fibrosis, the most extensive HFpEF endocardial biopsy study to date identified moderate to severe fibrosis in only 27% of patients[42], suggesting that additional factors beyond fibrosis contribute to ventricular wall stiffness in HFpEF

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