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The breadth of the pathways involved explains why BPC-157 is studied across gastrointestinal, musculoskeletal, and neurological recovery models

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The drug improves fluid absorption, which is the goal but the same effect drives the labeled risks of fluid overload and congestive heart failure if intravenous volumes are not stepped down, and of fluid and electrolyte imbalance if the drug is stopped abruptly while the mucosa regresses [1]