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Blockade of voltage dependent Na+ channels and increased brain levels of GABA are possibly the result of inhibition of GABA degradative enzymes or inhibition of GABA reuptake by neuronal cells, and are though to contribute to its anticonvulsant effect
By contrast, It activates ERR, ERR, and ERR, with the strongest reported potency at ERR
Approximately 12% of patients may also experience renal failure in the setting of acute tubular necrosis with or without hepatic necrosis
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