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Semaglutide activated the P38 MAPK signaling pathway To further explore the mechanism by which Semaglutide inhibits the progression of OSCC, we investigated the phosphorylation status of three MAPK signaling pathways (ERK1/2, SAPK/JNK and P38) in Cal27 and HSC4 cells after Semaglutide treatment (0, 5, or 10 mol/L, 48 h) by western blot analysis

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