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In terms of mechanisms, tesofensine inhibits the uptake of serotonin, noradrenaline, and dopamine, leading to appetite suppression and increased feelings of fullness

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Comparative metabolic and hepatic effects of GLP-1-based mono-, dual-, and triple-agonist therapies GLP-1 receptor signaling Intestinal L-cells serve as the primary source of GLP-1 synthesis, which operates via G-protein coupled receptors present in pancreatic beta cells, gastrointestinal and central neural systems, cardiac tissues, and kidneys [26]

Inactivation by EDTA or phenanthroline may be reversed by adding Zn 2+ but not Ca 2+ (Mllby and Wadstrom 1973)