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These disruptions lead to water and sodium influx, potassium efflux, cell swelling, membrane potential changes, and eventual cell lysis, which then releases intracellular contents, eliciting an inflammatory phenotype through DAMP release and triggering immune responses (Murao et al., 2021)

Cysteine residues can form disulfide bonds with each other or oxidize to cysteic acid

This antioxidant action helps reduce harmful compounds in liver cells, facilitating toxin removal and supporting tissue regeneration, especially valuable for those with liver inflammation

Approaches that inhibit glucose transporters (such as GLUT1 or GLUT3) offer upstream control of nutrient supply but often face compensatory upregulation of alternative transporters and may trigger systemic toxicity due to the essential role of glucose uptake in normal tissues