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These results collectively demonstrate that VPA exposure induces widespread microglial activation across key brain regions, and that hCiPSC-NSCs treatment effectively mitigates this neuroinflammatory response

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This enables us to examine the effect of GSH synthesis capacity on the ability of hepatocytes to detoxify NAPQI, the accumulation of NAPQI-induced liver damage, and the effects of different doses and timing of NAC in emergency departments

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