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MPMs were seeded in the upper chamber of the Transwell system with a pore size of 0.4 m and co-cultured with MCECs in the 6-well plate, followed by treatment with GHK-Cu (62.5 M)

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genes differentially expressed only with combination treatment) identified activation of p53 pathway, induction of apoptosis, and downregulation of MYC expression, thus functionally demonstrating restoration of wild-type p53 function

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