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In conclusion, we found that l-valine is a powerful stimulator of GLP-1 secretion both in rats and mice (ex vivo and in vivo), and we suggest a mechanism for l-valine stimulated GLP-1 release involving intracellular metabolism of the amino acid, leading to the closure of K ATP -channels, membrane depolarization and activation of V-gated Ca 2+ -channels (Fig

Our finding, in addition to being supported by those of previous studies [29-36], could explain why the cellular level of angiogenesis was significantly increased in ischemic zone in DPP4 D-HS and F344 HS + Sita groups than in F344 HS group