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To assess whether GLP-1(2836) can exert direct cardiac effects, we used an ex vivo isolated heart global IRI model under constant coronary perfusion pressure (4) (Figure 1D)

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Treatment of breast and ovarian cancer cells with the duel IGF-1R/IR tyrosine kinase inhibitor (TKI) BMS-536924 caused a reciprocal upregulation of the erbB family of receptors which conferred resistance to IGF-1R inhibition [48]