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As one medical commentary lamented, the literature on BPC-157 is sparse and fraught with methodological weaknesses, often relying on authors who have a vested interest in promoting the peptide

Tat upregulates lncRNA Xist, which sponges miR-124, leading to CCL2 overexpression and microglial activation [147]

Therefore, whether these immunomodulatory effects translate to a neuroprotective phenotype in the context of MS remains a critical open question that can only be addressed in disease-relevant preclinical models, such as Experimental Autoimmune Encephalomyelitis (EAE)