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The various genomic changes causing PWS all lead to loss of expression of the paternally expressed genes on chromosome 15q11.2q13 through loss or failure of expression because the maternal contribution has been programmed by epigenetic factors (eg, DNA methylation) to be silenced

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This can surely be called a landmark decision, which, as per health experts, could open up treatment access to more patients

Previously, a Phase 3 clinical trial for the amustaline/glutathione (GSH) PR system was halted when antibodies specific to acridine (a by-product of amustaline) were found in patients