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Adipose tissue in metabolic disease is itself an inflammatory organ, secreting cytokines (adipokines) that sustain the NF-B-driven inflammatory state that makes insulin resistance self-perpetuating

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By interacting with lectin-type Ox-LDL receptor-1, Ox-LDL is capable of suppressing the constitutive eNOS expression [107], elevating the expression of adhesion molecules and chemokines in ECs, and inducing macrophage proliferation, collagen production, vascular smooth muscle cell migration, and platelet activation [108]