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Additionally, mitochondria release apoptosis-inducing factor (AIF) and endonuclease G (Endo G), which mediate caspase-independent apoptosis (Taneja et al., 2001)

Testicular SOD activities that are largely confined to the seminiferous tubules did not change dramatically under these circumstances.95 It is therefore possible that the site of free radical generation in response to gonadotrophin withdrawal involves electron leakage from the inhibited steroidogenic pathway of the Leydig cells

5, C), Hixson-Crowell (R 2 = 0.91, Fig