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As mentioned above, within the IMM, cytochrome c participates in the mitochondrial ETC, using its heme group as a redox intermediate to shuttle electrons between complex III and complex IV

This form of resistance is generally associated with the accumulation of ROS and localized cell death in infected plant tissues

Recently, inhibitors targeting FA metabolism have been applied in preclinical studies [52], e.g., CD36 blockers are optionally combined with ICB therapy for melanoma and pancreatic cancer

Both SLC7A11 knockdown or overexpression can induce ferroptosis in hepatocytes of MAFLD model mice, but the mechanisms are different