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Remarkably, the vast majority of the molecules identified in this screen have been described to form complexes with metal ions or to either inhibit NF-B and/or the Akt/GSK3 pathway (Table 1)

Coincidently, earlier reports revealed the deposition of non-heme iron in the deep cortex and hippocampus in the postmortem brain tissues of mTBI patients ( Current Understanding of Secondary Brain Injury After Traumatic Brain Injury After TBI, the primary injury leads to neuronal death around the injury site

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And so, they apply to the joints, so the joint gets a little bit more attention at that time