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10.1016/s0928-4257(01)00040-7 83 Stancic-RokotovD.SlobodnjakZ.AralicaJ.AralicaG.PerovicD.StaresinicM.et al (2001b)

This special interplay further highlights that the loss of any one system can overwhelm the compensatory capacity of the others, forcing the CNS into a state of redox imbalance, glial activation, and progressive inflammation (Figure 1)

KPV is studied for anti-inflammatory activity, specifically NF-kB inhibition and preferential uptake in inflamed intestinal tissue via the PepT1 transporter

W odrnieniu od peptydw acylowanych (jak semaglutyd) BPC-157 nie ma przyczonego acucha kwasu tuszczowego jego stabilno wynika z samej sekwencji, nie z modyfikacji post-translacyjnej