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BPC-157 administration in rat models of mesenteric artery ligation has been shown to restore NO bioavailability in ischemic tissue beds within timeframes as short as 30 minutes post-administration, an effect blocked by pre-treatment with L-NAME (a non-selective NOS inhibitor), confirming the NO-dependent mechanism

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In turn, this inflammatory milieu, characterized by the production of inflammatory and pro-fibrotic cytokines, further exacerbates microvascular endothelial cell injury and apoptosis (174, 175), creating a vicious cycle that culminates in thrombosis, inflammation, and a myriad of clinical manifestations ranging from skin to parenchymal organs (174, 176)

Unger, Ketterer, Dupr, Eisentraut