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One study found that 10% muscle mass loss over 68 weeks on a GLP-1 equaled roughly two decades of normal age-related muscle decline

In neurons, cAMP and PKA signaling pathways regulate synaptic plasticity by regulating pathways related to N-Methyl-D-aspartic acid (NMDA) and aminomethylphosphonic acid receptor signaling such as calcium influx into intracellular membranes to change synaptic plasticity for long-term potentiation related to memory function [42-45]