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For example, mitochondrial dysfunction has been proposed as a source of increased oxidative stress in ASD [53] but within a distinct subgroup [54], or associated with co-occurring epilepsy [55]

This combination is believed to enhance the benefits of each peptide, providing cellular rejuvenation through telomere support and improved immune function

To circumvent the high lability of Cys acyclic disulfides during NCL and to use Cys thiol as a redox switch for controlling protein assembly, embedding the Cys thiol in a cyclic dichalcogenide represents a feasible solution as such species are known to be significantly less oxidizing than their linear counterparts 16

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