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Administration of MPTP or rotenone in mice promotes the development of mitochondrial dysfunction via inhibition of mitochondrial complex I, suggesting that inhibition of mitochondrial complex I and propagation of mitochondrial dysfunction could be the primary event in PD neuropathology

The biological mechanism linking folate and B 12 during pregnancy with offspring neurodevelopment is likely to be via the roles of these vitamins in OCM and the generation of SAM, which in turn is essential in donating methyl groups in numerous transmethylation reactions including the synthesis of neurotransmitters and DNA methylation (Reference Bailey, Stover and McNulty5,Reference Allen, Miller and De Groot7)