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Description

ER stress and unfolded protein response (UPR) signaling modulate GLP-1 receptor signaling in the pancreatic islets

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Dying for a cause: NETosis, mechanisms behind an antimicrobial cell death modality

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This limits the applicability of trial results to diverse clinical settings where patients often present with multiple health conditions

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Lancet 2 , 13001304 (1987)

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