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It has been demonstrated that the promotion of HDAC4 phosphorylation is found after overexpression of CDKL5 via facilitating the cytoplasmic retention of HDAC4 while loss of CDKL5 leads to the decrease of HDAC4 phosphorylation, ultimately increasing the translocation of HDAC4 into the nucleus [105]

Zhu S, Fisher LA, Bessho T, Peng A

In turn, these infected vesicles act as a Trojan horse when recognized and phagocytosed by macrophages, facilitating bacterial dissemination [45]