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In support, intravenous or intravitreous L-NAME application, given before, would aggravate the harmful effect of the major noxious procedure (i.e., ischemia by increasing intraocular pressure above systolic pressure by the infusion into the anterior chamber

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On the other hand, adipocytes subjected to oxidative stress express distress factors which are subsequently identified by the cluster of differentiation 8 (CD8), lymphocytes as well as natural killer cells (NK) which will synthesize interferon- (IFN-) inducing the transition of adipose tissue macrophages from the anti-inflammatory (M2) state to the pro-inflammatory (M1) state triggering inflammation [16]

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