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Beyond FSP1 there are therapeutic opportunities across the CoQ 10 oxidoreductase network While FSP1 currently has the strongest targetability signal, other CoQ 10 oxidoreductases may be therapeutically leveraged in defined genetic, metabolic or tissue contexts: (1) DHODH axis (mitochondrial defense in select contexts): DHODH connects pyrimidine synthesis to mitochondrial CoQ reduction, and in some settings, DHODH inhibition has been proposed to heighten ferroptosis sensitivity, especially in GPX4-low states
However, sulforaphanes origin in cancer research highlights one of its greatest strengthprevention
Moreover, BPC157 increases vessel density both in vivo and in vitro (in living organisms and in laboratory test tubes respectively) and speeds the process of recovery of blood flow in ischemic muscle tissue
See How to Read a Janoshik COA and the Lab Results page
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