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Third, elucidate the synergistic and/or antagonistic effects of the metabolites (e.g., 1,3-diaminopropane) on iron release during signaling pathways like HIF-2/FTN, yet verify their regulation of joint ferroptosis

!Copper chelators (penicillamine, trientine) - These medications remove copper from your bodythey would directly counteract GHK-Cu

Due to the cysteine residues of GSH, it can be readily oxidized to glutathione disulfide (GSSG) by electrophilic species directly (e.g., combine with NO to form less reactive S-nitrosoglutathione), or indirectly through enzymatic reactions (e.g., reduce hydrogen peroxide to water by glutathione peroxidase)