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It is, therefore, reasonable to consider that the postprandial rise in GLP-1 might affect feeding behavior after RYGB, and to a lesser extent SG, where the increase in GLP-1 is less marked (72) have shown that inhibition of GLP-1 actions in the postprandial period has limited effects on glucose concentrations in people after RYGB

In the rodent high fat fed male mice model, administration of semaglutide at the dose of 2800 g/kg/day resulted in a significant weight loss attributed to a decreased food intake [70]

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