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Our study demonstrates that cysteine-deficiency-induced CoA loss results in metabolic reprogramming and inefficiency, leading to urinary excretion of both glycolytic and tricarboxylic-acid-cycle intermediates, ketone bodies and other biosynthetic intermediates, contributing to the observed weight loss

& Kofler, B

Hell J Nucl Med 20(1):5156 Zimmermann MB, Galetti V (2015) Iodine intake as a risk factor for thyroid cancer: a comprehensive review of animal and human studies

NAD+ primarily functions as a coenzyme in energy metabolism and DNA repair, while glutathione acts as the body's master antioxidant for detoxification and cellular defense