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Excess ROS acts as a second messenger and regulates the function of important proteins, including Kelch-like ECH-associated protein 1 (Keap1), protein kinase C (PKC), and IB kinase by interacting with the cysteine residues (known as redox sensors) and causes their oxidation [10,25]

Liraglutide attenuates type 2 diabetes mellitus-associated non-alcoholic fatty liver disease by activating AMPK/ACC signaling and inhibiting ferroptosis

Beyond the Primary Endpoint: Cardiometabolic and GH-Axis Signals A fair comparison should acknowledge the secondary signals each peptide generated, without inflating them into approved benefits